EMF Blocking and EMF Shielding
If you reside in an apartment or a house or simply need to ensure that your house is free of electromagnetic fields There are a variety of methods to reduce exposure. One of the most effective is to reduce the use of electronic devices. You could also consider EMF block paint to prevent EMF radiation from reaching your house. emf blockers to shield your house against EMF radiations is to put up an RF shielding canopy. https://anotepad.com/notes/jke7hpwa is a cloth of net that contains EMF shielding and is used to block EMFs from entering a space. Another option is to get your home equipped with a conductive enclosure. These enclosures are known as Faraday cages.
Numerous studies have demonstrated that the non-ionizing RF EMF can cause antiproliferative effects in HCC cells. emf blocker that drives AM RF EMF's anticancer activity in vitro is believed be based on the downregulation in cancer-related stem cells. This may account for the long-term effects observed in some patients with advanced HCC. However, the mechanism behind AM EMF's effects on cancer patients is not clear.
Effects on the effects of AM electromagnetic fields (RFEM) on HCC tumour growth in vivo was studied in mice. The tumours were divided into 3 groups. First, the group that was unaffected to RF EMF. The second group was exposed RF EMF at a frequency that is similar to the one used by humans. Third group members were exposed RF EMF at HCC-specific modulation frequencies. The effect of HCCMF on the tumours was assessed against the effect of RCF. The results revealed that tumours treated with HCCMF had significant shrinkage. However, the tumors treated with RCF did not show any evidence of shrinkage of the tumor.
The mechanism behind tumor-specific AM RF EMF could be due to the fact that cancer cells require Cav3*2 voltage calcium channels for their proliferation and down-regulation. AM RF EMF's antiproliferative effects in HCC cells is controlled through CACNA1H the protein that is responsible for the influx of Ca2+ specific to tumours. The findings suggest that CACNA1H could have more broader implications in the treatment and diagnosis of different cancers.
The tumours in the controls were never exposed to RF EMF, and were fed a standard mouse diet. The tumours in HCCMF HCCMF group were infected with Huh7 cells when they were 5 to 7 weeks old. The tumors were then killed after they had a high burden.
The tumours in the three groups showed distinct growth curves. The tumours treated with HCCMF saw a significant reduction in size of the tumor after eight weeks. However, the tumours which were treated by RCF didn't show shrinkage. The difference was significant. The tumors treated with RCF were able to show necrosis, which is typical in tumors that have been exposed to RCF. There is a possibility that the necrosis is caused by an absence of oxygen in the more invasive tumours.
In conclusion, the findings indicate an AM-RF EMF exhibits anticancer activity in vitro and in the vivo. A number of studies have proven the fact that AM RF EMF produces measurable shrinkage of tumors in HCC patients. The possibility is that the AM EMF causes these effects through CACNA1H, a protein that is involved in the process of tissue-specific Ca2+ influx. In addition, AM RF EMF may cause a lasting effect on the growth of HCC tumors in the vivo.